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Pharmaco - Neuroimmunology
Pharmacodynamics
(As described in essentials of medical pharmacology by K. D. Tripathi)
Pharmacodynamics
is a study of dose- effect relationship or drug-drug interaction in vivo. Drugs do not impart new functions to any system, organ or cell; they only alter the pace of ongoing activity. The basic types of drug action at cellular level are stimulation, depression, irritation, replacement and cytotoxic action.
Pharmacokinetics
is quantative study of drug movements in, through and out of the body. If drug is administered systemically, there are two components in the dose-response relationship; dose-plasma concentration and plasma concentration-response relationship. Having kept all the standard parameters constant, the dose response relationship is never congruent in two individuals. This is explained on the basis of every patient's DNA-gene-sequence homology and their expression varies individual to individual and at a different time in the same individual. Chemical equivalence is not a bioequivalence.
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Scientific Analysis Of The Above Events
Pharmacokinetic parameters i.e. absorption, circulation and excretion in vivo is controlled by nervous system. Functions of nervous system are regulated by vital force energy. At the receptor level the drug-receptor works on the basis of expression of c-DNA genes. (Refer: Future trends in Pharmacodynamics by K. D. Tripathi). Genetic expression is a function of bioenergy. It is as clear as sunlight; therapeutic outcome of drugs is decided by vital force energy. Hence optimization of vital force energy gives desired therapeutic results and minimum adverse effects. (Fig. No. 1)
Drug is a chemical compound. After administration of drug to a patient it undergoes the various stages; dissolution and disintegration. As a result binding energy is released, which interacts with patients' bioenergy and resultant is either therapeutic or non-therapeutic effect (adverse drug reaction)
Fig 1
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When Neuroimmunisation Therapy is combined with other therapy (drug + Panaceand), it optimizes bioenergies for the effects of the drugs and it results into enhancement of the therapeutic effect and minimization of adverse effects. (Fig. 2).
Fig.2
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